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The following is a summary of “Apelinergic System in the Left Ventricle Adverse Remodeling After Myocardial Infarction: A ...
When the workload on the heart increases, the ventricular wall may thicken too, known as cardiac hypertrophy. This is an adaptive response that reduces pressure on the heart and maintains the activity ...
Now, a study published in the journal Cellular and Molecular Life Sciences has identified a new factor linked to the development of pathological hypertrophy. The results of the study suggest that ...
When the workload on the heart increases, the ventricular wall may thicken too, known as cardiac hypertrophy. This is an ...
Recent FDA updates in the areas of cardiac intervention, hypertension and arrhythmia disorders expand the pool of available ...
Adolescent hyperglycemia and worsening insulin resistance are associated with an increased risk for worsening structural and functional cardiac damage.
At four years' follow-up, 21% of the lamin arms and 6% of the DCMwt arm had experienced MACE. Univariable Cox regression showed that in the lamin arms, each 1% increase in global LGE and each 1% ...
Cardiac remodeling is a process leading to changes in size, architecture, and histology of the heart. It occurs in myocardial infarction, valvular disease, arrhythmias, acute and chronic myocarditis, ...
Heart failure is associated with a high rate of mortality and morbidity, and ventricular remodeling invariably precedes heart failure. Ventricular remodeling is fundamentally driven by ...
and right ventricular remodeling is present in patients with uncomplicated diabetes or prediabetes. 20,21 Potential contributing mechanisms include promotion of myocardial fibrosis, inflammation ...
Within the last 24 months, I have financial relationship(s) or affiliation(s) with a manufacturer, marketer, reseller, or distributor of a healthcare product or service involved in the management of ...
Secondary outcomes included HF hospitalization, cardiac death, all-cause death, significant ventricular arrhythmia and evidence of cardiac reverse remodeling such as change in LVEF during follow-up.